Analysis of the partial 16S rRNA gene sequences of HQ1 and HQ2 resulted in low levels of homology (96%) to type strains of H. haemolyticus (CIP 103290; GenBank accession no.

Phylogenetic trees constructed on the phylogeny.fr platform showing the relatedness of seven invasive Haemophilus quentini clinical isolates from Ontario, Canada (HQ1 to HQ7), clinical H. quentini isolates from Hong Kong (HK-1) and the United Kingdom (MP1), H. haemolyticus reference (M19346) and type (ATCC 33390T) strains, two nontypeable H. influenzae biotype IV clinical isolates from Ontario, Canada (NTHi1, NTHi2), and an H. influenzae reference strain (Rd KW20) based on hpd (A), ompP6 (B), sodC (C), the 16S rRNA gene (D), and concatenated sequences of six MLST genes (adk, atpG, frdB, mdh, pgi, and recA) (E). Of the 96 liveborn neonates, 4 (4%) subsequently died (Table 3) and 1 of 10 bacteraemic neonates died. Histopathological material was re‐reviewed by a perinatal pathologist not blinded to the original diagnosis. Strains of NTHi associated with vulvovaginitis have been found to be predominantly biotype II and strains associated with obstetric sepsis have been found to be biotype II or III in previous studies.21, 24 Comparing the incidence and biotype composition of oropharyngeal carriage of NTHi in mothers affected by obstetric NTHi would be of interest. We present a case of H, influenzae biotype IV infection in a premature (32 weeks) neonate. Two women had NTHi isolated during different pregnancies. Based on the sodC gene, HQ1 to HQ6 were split into two groups: HQ1, HQ2, and HQ5 were identical as a group, while HQ3, HQ4, and HQ6 were identical as the second group.

Ninety‐six babies were liveborn. Additionally, bexA-specific PCR confirmed the absence of the capsule transport gene in all the H. quentini isolates.

Antimicrobial resistance patterns of Haemophilus influenzae isolated from patients with meningitis in São Paulo, Brazil. The remaining adult female patient was an elderly woman with an H. quentini bloodstream infection (previously reported by Eshaghi et al.

lab icon. Summary: Neonatal infection due to Haemophilus influenzae has several clinical similarities to infection by the more common Streptococcus agalactiae (Strep group B). Please log in. Characteristics of seven invasive clinical isolates of suspected Haemophilus quentini from Ontario, Canada. Thirty‐one of 33 placentas sent for histology demonstrated features of chorioamnionitis.

Rev. HQ7 differed from HQ1, HQ2, HQ5, and HK-1 by 12 SNPs (96% similarity), from HQ3, HQ4, and HQ6 by 10 SNPs (96.7% similarity), and from MP1 by 14 SNPs (95.3% similarity). Four liveborn neonates died (4%). Su genoma consta de 1.830.140 pares de bases y contiene 1.740 genes.[2]​. Most placental samples cultured grew NTHi as the only identified pathogen. The significance of this pathogen may be underestimated due in part to difficulties with identification, as well as confusion with the changing nomenclature.



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