Human genetic variability has been recognised to be of great relevance in host responses to Mycobacterium tuberculosis infection and in regulating both the establishment and the progression of the disease. It is caused by the gram-positive acid-fast bacilli Mycobacterium tuberculosis. 2020 Mar;92:21-28. doi: 10.1016/j.ijid.2019.12.013. 2019 Aug 27;7:350. doi: 10.3389/fped.2019.00350. Tuberculosis is a disease caused by the bacterium Mycobacterium tuberculosis. [1][2] As is common in GWAS, the variants discovered have moderate effect sizes - but have shown that TB has high polygenic heritability. Articles in English retrieved and relevant references cited in these articles were reviewed. Information provided on this site is for informational purposes only; it is not intended as a substitute for advice from your own medical team. Learn how and when to remove this template message, "Common variants at 11p13 are associated with susceptibility to tuberculosis", "Genome-wide association analyses identifies a susceptibility locus for tuberculosis on chromosome 18q11.2", "Early progression to active tuberculosis is a highly heritable trait driven by 3q23 in Peruvians", https://en.wikipedia.org/w/index.php?title=Genetics_of_tuberculosis&oldid=961126634, Articles with too few wikilinks from December 2018, Articles covered by WikiProject Wikify from December 2018, All articles covered by WikiProject Wikify, Creative Commons Attribution-ShareAlike License, This page was last edited on 6 June 2020, at 19:04. Impact of Host Genetics and Biological Response Modifiers on Respiratory Tract Infections. | It is a bacterial infection, not a genetic disease. Genome-wide associations identify novel candidate loci associated with genetic susceptibility to tuberculosis in wild boar. [3] These studies suggest that while only a handful of alleles have been found, larger well designed studies may find more. eCollection 2019. Copyright © 2014 Health Grades Inc. All rights reserved. candidate gene studies; estudios de asociación de genomas completos; estudios de genes candidatos; genome-wide association studies; genética humana; génétique humaine; human genetics; tuberculose; tuberculosis; études d'association du génome entier; études de gènes candidats. Lacoma A, Mateo L, Blanco I, Méndez MJ, Rodrigo C, Latorre I, Villar-Hernandez R, Domínguez J, Prat C. Front Immunol. Our servers have detected that you are accessing this site from a country that is a member of the European Union. Int J Infect Dis. Clipboard, Search History, and several other advanced features are temporarily unavailable.
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